Skip Navigation LinksHome > Explorations > Grants Awarded

 Grand Challenges Explorations Grants

Grand Challenges Explorations fosters creative projects that show great promise to improve the health of people in the developing world. Initial grants are awarded two times a year, and successful projects have the opportunity to receive additional funding of up to $1 million.

On May 10, 2010, the Gates Foundation announced that 78 new global health projects received Grand Challenges Explorations grants. Learn more about these below. Round 5 grants will be announced in October 2010.

To review all 340 Explorations projects, select "Show All Rounds" in the Round drop-down menu.

 Find Awarded Grants By

Topic
Technologies
Round
Showing Grants 1 to 10 of 13
A New Way to Prevent HIV Infection During Breastfeeding
Primary Investigator:
David Sokal, Family Health International, Durham, NC, United States - US
Topic:
Round:
Round 2 – May 2009
David Sokal of Family Health International in the U.S., with colleagues at Cambridge and Drexel Universities, will develop and test low-cost filters coated with safe microbicides that can be inserted into tips of nipple shields to prevent HIV transmission during breastfeeding.
A Non-Pathogenic Chimeric THLV-1/HIV-1 Viral Genome as a Model to Study Superinfection Restriction
Primary Investigator:
Kuan-Teh Jeang, National Institutes of Health, Bethesda, MD, United States - US
Topic:
Round:
Round 2 – May 2009

Kuan-Teh Jeang of the National Institutes of Health in the U.S. will investigate whether cells infected by one virus become resistant to infection from other viruses, and if this viral interference can confer protection against HIV. The team will develop an attenuated virus to test whether over-expression of viral envelope proteins within cells can confer resistance to further HIV infection.

A Novel Therapeutic Strategy to Control HIV-1 Infection
Primary Investigator:
Qugui Yu, Indiana University, Indianaoplis, IN, United States - US
Topic:
Round:
Round 2 – May 2009
The complement system, part of the innate human immune reaction which helps clear viruses and other pathogens, is thought to be inactivated by HIV. Qigui Yu of Indiana University School of Medicine in U.S. will attempt to reverse this inactivation in an effort to render HIV and infected cells susceptible to complement-mediated attack.
A Small Molecule That Blocks Male-to-Female Sexual Transmission of HIV
Primary Investigator:
David Eisenberg, University of California, Los Angeles, CA, United States - US
Topic:
Round:
Round 2 – May 2009

Recent evidence suggests that HIV infection may be drastically enhanced when a specific protein found in human semen is present in fibril form. David Eisenberg of UCLA in the U.S. will design and test a small peptide that can effectively block formation of fibrils on this protein. If successful, the therapy could be administered via spray or liquid drops to inhibit transmission of HIV.

Breastmilk shield to prevent HIV transmission
Primary Investigator:
Gadi Borkow, Cupron Inc., Greensboro, NC, United States - US
Topic:
Round:
Round 2 – May 2009
Gadi Borkow of Cupron, Inc. in the U.S. will study the efficacy of using newly developed copper-oxide based filters that deactivate a wide range of viruses, including HIV-1, as a shield to enable HIV-infected mothers to breastfeed their infants without risking transmission of the virus.
HIV Protease-Dependent Activation of a Cytotoxic Prodrug
Primary Investigator:
Craig Crews, Yale University, New Haven, CT, United States - US
Topic:
Round:
Round 2 – May 2009
For viral replication, HIV viruses are dependent upon proteins, called proteases, to appropriately cleave peptides and form functional viral particles.  Craig Crews of Yale University in the U.S. will attempt to exploit these proteases by designing a drug that will cleave only to HIV protease and release a cytotoxin that results in programmed cell death.
Immunotherapy with iPS Derived From HIV-1 Specific B-Cells
Primary Investigator:
Irvin Chen, University of California, Los Angeles, CA, United States - US
Topic:
Round:
Round 2 – May 2009

HIV destroys helper T cells, which are essential to activation of B-cells. Irvin Chen of UCLA in the U.S. will utilize inducible pluripotent stem cell technology to generate a constant, self-renewing source of antigen-specific B-cells, which target conserved HIV epitopes to eliminate HIV-infected cells.

Mortalizing HIV – A Novel Method to Help Eradicate HIV
Primary Investigator:
Reuben Harris, University of Minnesota, Minneapolis, MN, United States - US
Topic:
Round:
Round 2 – May 2009
A high HIV mutation rate enables escape from powerful immune responses and anti-retroviral drugs. Reuben Harris of the University of Minnesota in the U.S. will test the hypothesis that HIV requires the human APOBEC3G protein to maintain a high mutation rate necessary for HIV survival. Inhibiting this protein may slow the mutation rate and make the virus more susceptible to immune responses.
Novel HIV-1 Env Immunogens for Immuno-Focusing
Primary Investigator:
Ruth Ruprecht, Dana-Farber Cancer Institute, Boston, MA, United States - US
Topic:
Round:
Round 2 – May 2009
Ruth Ruprecht of the Dana-Farber Cancer Institute in the U.S. will develop a new vaccine platform for HIV based on the hypothesis that immunodominant regions of the virus may be irrelevant to neutralizing the virus and also prevent access to neutralizing epitopes in conserved regions. The team will also use structural mimics of important epitopes in an effort to generate a strong, broadly neutralizing antibody response against these conversed sites. 
Prevent HIV Infection by Naturally Occurring Antivirals
Primary Investigator:
John Fahey, Darmouth College, Hanover, NH, United States - US
Topic:
Round:
Round 2 – May 2009
To harness a woman’s immune system to prevent HIV-1 infection, John Fahey and Charles Wira at Dartmouth Medical School in the U.S. will identify SERMs (selective estrogen receptor modulators) similar to those used for treating breast cancer and osteoporosis that can induce local immune protection in the reproductive tract against HIV without compromising normal reproductive function or increasing the risk of HIV infection.
1 - 10 Next

 How to Apply

 Map of Grants